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Insulin secretion was only affected by somatostatin at 12 mmol/l glucose, demonstrating that a tonic inhibition by endogenous somatostatin does not contribute to the very low levels of insulin at low glucose levels, whereas a paracrine inhibitory effect by local somatostatin plays a significant role in the regulation of insulin secretion at higher glucose levels

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Its defining pharmacological feature is negligible cortisol and prolactin co-stimulation at GH-stimulating doses, making it the preferred compound in research protocols where isolated GH-axis stimulation is required

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